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When Daniel developed his
worst autistic symptoms he also developed
Vitiligo. At the time I didn't know what
was the relationship between the Vitiligo and
the autistic symptoms. We performed many
immune panel (Lymphocyte Subset Panel 1) lab
tests. At first, Daniel's immune panel
looked normal to the naked eye. Once we
got the February 2008 brain SPECT scan results
back showing temporal lobe hypoperfusion caused
by brain inflammation we decided to start
treating Daniel with antiviral medications.
Interesting enough, after
the first day of the antiviral therapy, Daniel's
neck lymph nodes became very enlarged.
They were a bit larger than a golf ball.
He was only 3 1/2 years old, so the lymph nodes
looked very big compared to his toddler neck.
After some research I learned that one of the
most common causes for neck lymph node
enlargement is viral infections.
After looking the lab test
results over and over, I noticed that the one
marker that was always high was the CD19+ cells.
When CD19+ cell levels are elevated it is
referred as CD19+ cell overexpression.
CD19+ cell overexpression is
known to induce autoimmunity.
Autoimmunity is the failure of an
organism to recognize its own constituent parts
as self, which allows an immune response
against its own cells and tissues. Any disease
that results from such an aberrant immune
response is termed an
autoimmune disease.
Vitiligo is associated with
autoimmune and
inflammatory diseases.
Once we started
treating
Daniel's viral infection, inflammation and
started regulating his immune system, we noticed
that the CD19+ cells count started to come down.
Once the CD19+ cell count started to go down,
Daniel's vitilgo started getting better.
Once the CD19+ cells were completely regulated,
the skin areas affected by the Vitiligo regained
its pigmentation. Daniel's autistic
symptoms became milder at the same rate as the
Vitiligo cleared up. On February 2009, we
did the second brain SPECT scan and the
radiologist was impressed and said that there
was only residual brain inflammation compared to
the brain inflammation detected in the
first brain SPECT scan.
B Cells Can Act Alone In Autoimmune Disease,
Yale Researchers Report
Daniel showed he suffered
from
Hypogammaglobulinemia
through two IGG lab tests.
Hypogammaglobulinemia is a type of
immune disorder characterized by a reduction
in all types of
gamma globulins. The immune panels
(Lymphocyte Subset Panel 1) lab tests
demonstrated that Daniel suffered from
autoimmunity due to the constant CD19+ (B Cells)
over expression. This confirmed
Daniel's autoimmune disorder diagnosis.
Immune Panel
Daniel's immune system was
very deregulated.
There were clear
signs of autoimmunity. After medical
treatment we were able to regulate his immune
system to almost complete proper function.
Immunoglobulins Panel In May
21, 2007 we test Daniel's (35 months old at the
time) immunoglobulin levels. His Immunoglobulin
G count came low which put him under the
diagnosis of hypogammaglobulinemic.
Hypogammaglobulinemia is a type of
immune disorder characterized by a reduction
in all types of
gamma globulins.
2 1/2 years later with the current medical
treatment his immunoglobulin levels are much
better which means that his immune system is
more capable of fighting infections.
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